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Fournisseur: Thermo Fisher Scientific
Description: Boc-Arg-OH 98%
Numéro de catalogue: (BOSSBS-13627R-CY3)
Fournisseur: Bioss
Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM: 1 * 100 µl


Fournisseur: Apollo Scientific
Description: Boc-Arg(NO₂)-OH

Numéro de catalogue: (BOSSBS-13627R-A750)
Fournisseur: Bioss
Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter?s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM: 1 * 100 µl


Numéro de catalogue: (BOSSBS-13627R-CY7)
Fournisseur: Bioss
Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM: 1 * 100 µl


Fournisseur: Apollo Scientific
Description: H-Arg-AMC·2HCl

Fournisseur: Apollo Scientific
Description: Boc-ARG(PMC)-OH 99%

Fournisseur: Thermo Fisher Scientific
Description: Fmoc-Arg(Boc)₂-OH 95%
Fournisseur: Thermo Fisher Scientific
Description: Boc-Arg(NO₂)-OH 98%
Numéro de catalogue: (ENZOBMLP1350025)
Fournisseur: ENZO LIFE SCIENCES
Description: Substrate for cathepsin H and aminopeptidases
UOM: 1 * 25 mg


Numéro de catalogue: (USBI130415)
Fournisseur: US Biological
Description: Anti-NLRP3 Mouse Polyclonal Antibody
UOM: 1 * 50 µG


Numéro de catalogue: (H62515.06)
Fournisseur: Thermo Fisher Scientific
Description: Boc-Arg(Mtr)-OH 98%
UOM: 1 * 5 g


Numéro de catalogue: (USBI130416)
Fournisseur: US Biological
Description: Anti-NLRP3 Rabbit Polyclonal Antibody
UOM: 1 * 1 EA


Numéro de catalogue: (USBI150280)
Fournisseur: US Biological
Description: Anti-NLRP3 Goat Polyclonal Antibody
UOM: 1 * 1 EA


Numéro de catalogue: (BOSSBS-13627R-A647)
Fournisseur: Bioss
Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM: 1 * 100 µl


Numéro de catalogue: (BOSSBS-13627R-HRP)
Fournisseur: Bioss
Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM: 1 * 100 µl


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