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Image non disponible-8.08913.5000

Tétrachlorure de zirconium pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Tétrachlorure de zirconium pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-8.14353.5000

Potassium hydroxyde pour la synthèse

Fournisseur: Merck
Description: Potassium hydroxyde pour la synthèse

MSMD

Image non disponible-8.01804.9068

Chlorure de benzoyle pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Chlorure de benzoyle pour la synthèse, Sigma-Aldrich®

MSMD

Image du produit-K1632

RevertAid™ H minus first strand cDNA synthesis kits

Fournisseur: Thermo Fisher Scientific
Description: RevertAid™ H Minus First Strand cDNA Synthesis Kit is a complete system for efficient synthesis of first strand cDNA from RNA templates. The kit includes RevertAid™ H Minus Reverse Transcriptase, which has a point mutation that completely eliminates RNase H activity. Therefore, it does not degrade RNA in RNA-DNA hybrids during synthesis of the first strand cDNA.

Image non disponible-8.03052.0005

Chlorure de diméthylammonium pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Chlorure de diméthylammonium pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-8.20293.0500

Chloroacétate de sodium pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Chloroacétate de sodium pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-8.02898.0100

Carbonate de diéthyle pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Carbonate de diéthyle pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-8.20611.9050

Carbonate de guanidinium pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Carbonate de guanidinium pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-BNUB1008-100

Anti-FUT3 Mouse Monoclonal Antibody (Purified) [clone: 2-25LE]

Fournisseur: Biotium
Description: The Lewis histo-blood group system comprises a set of fucosylated glycosphingolipids that are synthesized by exocrine epithelial cells and circulate in body fluids. The glycosphingolipids function in embryogenesis, tissue differentiation, tumor metastasis, inflammation, and bacterial adhesion. They are secondarily absorbed to red blood cells giving rise to their Lewis phenotype. This gene is a member of the fucosyltransferase family, which catalyzes the addition of fucose to precursor polysaccharides in the last step of Lewis antigen biosynthesis. It encodes an enzyme with alpha(1,3)-fucosyltransferase and alpha(1,4)-fucosyltransferase activities. Lewis blood group antigens are carbohydrate moieties structurally integrated in mucous secretions. Lewis antigen system alterations have been described in gastric carcinoma and associated lesions. Anomalous expression of Lewis B antigen has been found in some non-secretory gastric carcinomas and colorectal cancers.

Image non disponible-BNUM1008-50

Anti-FUT3 Mouse Monoclonal Antibody (Purified, BSA-free) [clone: 2-25LE]

Numéro de catalogue: (BNUM1008-50)
Fournisseur: Biotium
Description: The Lewis histo-blood group system comprises a set of fucosylated glycosphingolipids that are synthesized by exocrine epithelial cells and circulate in body fluids. The glycosphingolipids function in embryogenesis, tissue differentiation, tumor metastasis, inflammation, and bacterial adhesion. They are secondarily absorbed to red blood cells giving rise to their Lewis phenotype. This gene is a member of the fucosyltransferase family, which catalyzes the addition of fucose to precursor polysaccharides in the last step of Lewis antigen biosynthesis. It encodes an enzyme with alpha(1,3)-fucosyltransferase and alpha(1,4)-fucosyltransferase activities. Lewis blood group antigens are carbohydrate moieties structurally integrated in mucous secretions. Lewis antigen system alterations have been described in gastric carcinoma and associated lesions. Anomalous expression of Lewis B antigen has been found in some non-secretory gastric carcinomas and colorectal cancers.
UOM: 1 * 50 µl
Image non disponible-8.21178.0250

Chlorure de trimethylammonium pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Chlorure de trimethylammonium pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-BOSSBS-12179R-CY5

Anti-KIR5.1 Rabbit Polyclonal Antibody (Cy5®)

Numéro de catalogue: (BOSSBS-12179R-CY5)
Fournisseur: Bioss
Description: The KIR family of potassium channels possess a greater tendency to allow potassium to flow into the cell rather than out of it. Kir4.1, also known as Kir1.2, is highly expressed in brain including glial cells, astrocytes and cortical neurons. Kir4.1 is also expressed in myelin-synthesizing oligodendrocytes and is crucial to myelination in the developing nervous system. The gene encoding human Kir4.1 maps to chromosome 1. Kir4.2, also known as Kir1.3, is expressed in kidney, lung, heart, thymus and thyroid during development. The gene encoding human Kir4.2 maps to chromosome 21 in the Down syndrome chromosome region 1, and Kir4.2 may play a role in the pathogenesis of Down’s syndrome. Kir 5.1 forms functional channels only by coexpression with either Kir4.1 or Kir4.2 in the kidney and pancreas. The gene encoding human Kir5.1 maps to chromosome 17.
UOM: 1 * 100 µl
Image non disponible-BOSSBS-12180R

Anti-KIR5.1 Rabbit Polyclonal Antibody

Numéro de catalogue: (BOSSBS-12180R)
Fournisseur: Bioss
Description: The KIR family of potassium channels possess a greater tendency to allow potassium to flow into the cell rather than out of it. Kir4.1, also known as Kir1.2, is highly expressed in brain including glial cells, astrocytes and cortical neurons. Kir4.1 is also expressed in myelin-synthesizing oligodendrocytes and is crucial to myelination in the developing nervous system. The gene encoding human Kir4.1 maps to chromosome 1. Kir4.2, also known as Kir1.3, is expressed in kidney, lung, heart, thymus and thyroid during development. The gene encoding human Kir4.2 maps to chromosome 21 in the Down syndrome chromosome region 1, and Kir4.2 may play a role in the pathogenesis of Down’s syndrome. Kir 5.1 forms functional channels only by coexpression with either Kir4.1 or Kir4.2 in the kidney and pancreas. The gene encoding human Kir5.1 maps to chromosome 17.
UOM: 1 * 100 µl
Image non disponible-BOSSBS-12180R-CY5

Anti-KIR5.1 Rabbit Polyclonal Antibody (Cy5®)

Numéro de catalogue: (BOSSBS-12180R-CY5)
Fournisseur: Bioss
Description: The KIR family of potassium channels possess a greater tendency to allow potassium to flow into the cell rather than out of it. Kir4.1, also known as Kir1.2, is highly expressed in brain including glial cells, astrocytes and cortical neurons. Kir4.1 is also expressed in myelin-synthesizing oligodendrocytes and is crucial to myelination in the developing nervous system. The gene encoding human Kir4.1 maps to chromosome 1. Kir4.2, also known as Kir1.3, is expressed in kidney, lung, heart, thymus and thyroid during development. The gene encoding human Kir4.2 maps to chromosome 21 in the Down syndrome chromosome region 1, and Kir4.2 may play a role in the pathogenesis of Down’s syndrome. Kir 5.1 forms functional channels only by coexpression with either Kir4.1 or Kir4.2 in the kidney and pancreas. The gene encoding human Kir5.1 maps to chromosome 17.
UOM: 1 * 100 µl
Image non disponible-8.14733.0500

Magnésium chlorure pour la synthèse, Sigma-Aldrich®

Fournisseur: Merck
Description: Magnésium chlorure pour la synthèse, Sigma-Aldrich®

MSMD

Image non disponible-BOSSBS-2076R-A555

Anti-Methamphetamine Rabbit Polyclonal Antibody (Alexa Fluor® 555)

Numéro de catalogue: (BOSSBS-2076R-A555)
Fournisseur: Bioss
Description: Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.
UOM: 1 * 100 µl
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