Votre recherche pour: [Arg+8]-Vasopressin+Acetate

Numéro de catalogue: (H62515.06)

Fournisseur: Thermo Fisher Scientific

Description: Boc-Arg(Mtr)-OH 98%

UOM: 1 * 5 g

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Numéro de catalogue: (BOSSBS-13627R-A647)

Fournisseur: Bioss

Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.

UOM: 1 * 100 µl

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Numéro de catalogue: (BOSSBS-13627R-HRP)

Fournisseur: Bioss

Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.

UOM: 1 * 100 µl

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Numéro de catalogue: (BOSSBS-13627R-A350)

Fournisseur: Bioss

Description: The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.

UOM: 1 * 100 µl

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Fournisseur: TCI

Description: Boc-D-Arg-OH·HCl·H2O ≥98.0% (sur base azote total)

Fournisseur: TCI

Description: Fmoc-D-Arg(Pbf)-OH ≥98.0% (par HPLC, analyse par titration)

Fournisseur: Apollo Scientific

Description: Fmoc-L-Arg(Me)2(Pbf)-OH

Numéro de catalogue: (PRSI25-909)

Fournisseur: ProSci Inc.

Description: SLCO3A1 mediates the Na+-independent transport of organic anions such as estrone-3-sulfate. It mediates transport of prostaglandins (PG) E1 and E2, thyroxine (T4), deltorphin II, BQ-123 and vasopressin, but not DPDPE (a derivative of enkephalin lacking an N-terminal tyrosine residue), estrone-3-sulfate, taurocholate, digoxin nor DHEAS.

UOM: 1 * 50 µG

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Fournisseur: TCI

Description: Fmoc-Arg(Pbf)-OH ≥98.0% (par HPLC, analyse par titration)

Fournisseur: Merck

Description: L(+)-Arginine pour biochimie, Millipore®

MSMD

Numéro de catalogue: (R8276-1MG)

Fournisseur: SIGMA ALDRICH MICROSCOPY

Description: Substrate for human renin, an aspartic protease which plays an important role in blood-pressure regulation.

UOM: 1 * 1 mg

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Numéro de catalogue: (SRE0003-10KU)

Fournisseur: SIGMA ALDRICH MICROSCOPY

Description: Serine protease that selectively cleaves Arg-Gly bonds in fibrinogen to form fibrin and fibrinopeptides A and B.

UOM: 1 * 1 EA

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Numéro de catalogue: (ENZOBMLAW97850005)

Fournisseur: ENZO LIFE SCIENCES

Description: This fluorogenic peptide is often the substrate of choice for assaying the trypsin-like activity of purified proteasomes. Benefits include a low Km, high specific activity, and the fact that the substrate is cleaved exclusively at the R-AMC bond.

UOM: 1 * 5 mg

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Numéro de catalogue: (BOSSBS-0841R)

Fournisseur: Bioss

Description: Glucagon plays a key role in glucose metabolism and homeostasis. Regulates blood glucose by increasing gluconeogenesis and decreasing glycolysis. A counterregulatory hormone of insulin, raises plasma glucose levels in response to insulin-induced hypoglycemia. Plays an important role in initiating and maintaining hyperglycemic conditions in diabetes. GLP-1 is a potent stimulator of glucose-dependent insulin release. Play important roles on gastric motility and the suppression of plasma glucagon levels. May be involved in the suppression of satiety and stimulation of glucose disposal in peripheral tissues, independent of the actions of insulin. Have growth-promoting activities on intestinal epithelium. May also regulate the hypothalamic pituitary axis (HPA) via effects on LH, TSH, CRH, oxytocin, and vasopressin secretion. Increases islet mass through stimulation of islet neogenesis and pancreatic beta cell proliferation. Inhibits beta cell apoptosis. GLP-2 stimulates intestinal growth and up-regulates villus height in the small intestine, concomitant with increased crypt cell proliferation and decreased enterocyte apoptosis. The gastrointestinal tract, from the stomach to the colon is the principal target for GLP-2 action. Plays a key role in nutrient homeostasis, enhancing nutrient assimilation through enhanced gastrointestinal function, as well as increasing nutrient disposal. Stimulates intestinal glucose transport and decreases mucosal permeability. Oxyntomodulin significantly reduces food intake. Inhibits gastric emptying in humans. Suppression of gastric emptying may lead to increased gastric distension, which may contribute to satiety by causing a sensation of fullness. Glicentin may modulate gastric acid secretion and the gastro-pyloro-duodenal activity. May play an important role in intestinal mucosal growth in the early period of life.

UOM: 1 * 100 µl

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Numéro de catalogue: (ENZOADI900058)

Fournisseur: ENZO LIFE SCIENCES

Description: For the quantitative determination of Complement C3a des arg in human EDTA plasma.

UOM: 1 * 1 KIT

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Numéro de catalogue: (ENZOBMLAM1010001)

Fournisseur: ENZO LIFE SCIENCES

Description: Negative control for integrin avß3 antagonist Cyclo [Arg-Gly-Asp-D-Phe-Val].

UOM: 1 * 1 mg

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